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Clinical Study Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc 2013

Different oxygen treatment pressures alter inflammatory gene expression in human endothelial cells.

Kendall AC, Whatmore JL, Harries LW, Winyard PG, Eggleton P, Smerdon GR — Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc, 2013

Tier 2, Indexed

Automatically imported from PubMed based on relevance criteria.

Summary

What Researchers Did

Researchers investigated how different oxygen treatment pressures (normobaric and hyperbaric) affected inflammatory gene expression in human endothelial cells under chronic wound conditions.

What They Found

Researchers analyzed the mRNA expression of 92 inflammatory genes. They found changes in genes related to adhesion molecules, angiogenesis, tissue remodeling, and cellular oxygen responses, with notable differences in expression observed between the various oxygen treatment pressures.

What This Means for Canadian Patients

This research suggests that the specific pressure used during oxygen therapy for chronic wounds could significantly impact treatment effectiveness by altering inflammatory responses at a cellular level. Optimizing oxygen treatment protocols based on these findings may lead to more effective wound healing strategies for Canadian patients.

Canadian Relevance

This study has no direct Canadian connection.

Study Limitations

This in vitro study on endothelial cells requires further research to translate its findings into optimized clinical protocols for chronic wound treatment.

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Study Details

Study Type Clinical Study
Category Wound Care
Source Pubmed
PubMed ID 23682543
Year Published 2013
Journal Undersea & hyperbaric medicine : journal of the Undersea and Hyperbaric Medical Society, Inc
MeSH Terms Apoptosis; Atmospheric Pressure; Cell Hypoxia; Chronic Disease; Diabetic Foot; Fibronectins; Gene Expression; Gene Expression Profiling; Human Umbilical Vein Endothelial Cells; Humans; Hyperbaric Oxygenation; Inflammation Mediators; Neovascularization, Physiologic; Oxidation-Reduction; Platelet Endothelial Cell Adhesion Molecule-1

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Disclaimer: This study summary is provided for informational and educational purposes only. It does not constitute medical advice. The information presented reflects the findings of the original research authors and may not represent the views of Canada Hyperbarics. Always consult a qualified healthcare professional before making treatment decisions.